氧化条件下巨噬细胞中蛋白质S-谷胱甘肽修饰的改变 （Guo et al., 2021）

蛋白S-谷胱甘肽化状态变化影响肺部疾病（Chia et al., 2020）

1.Decreased Expression of the Host Long-Noncoding RNA-GM Facilitates Viral Escape by Inhibiting the Kinase activity TBK1 via S-glutathionylation （IF= 43.474, Q1, Immunity） S-谷胱甘肽化调TBK1 活性酶并损害先天畸形免疫性和病菌肇事

2.Oxidative stress-induced FABP5 S-glutathionylation protects against acute lung injury by suppressing inflammation in macrophages （IF=16.6，Q1，Nature communication） 硫化应激状态诱发的 FABP5 S-谷胱甘肽化在抑止巨噬癌细胞炎症病变来应对发生急性肺磨损

3.S-glutathionylation of Hsp90 enhances its degradation and correlates with favorable prognosis of breast cancer （IF=10.787，Q1，Redox Biol） Hsp90表达出及谷胱甘肽化环境能作为恶性肿瘤的疗效生物体标志牌物或开展靶点

4.Protein S-glutathionylation stimulate adipogenesis by stabilizing C/EBPβ in 3T3L1 cells (IF=5.834, Q1, FASEB J)（注可册除） 血清质S-谷胱甘肽化借助增强3T3L1肿瘤细胞中的 C/EBPβ伤害性人体脂肪制成